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Heart Research
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Heart Research- Slides

Exercise training improves function of circulating angiogenic cells in patients with chronic heart failure.
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Alterations in circulating angiogenic cells (CAC) and endothelial progenitor cells (EPC) known to contribute to endothelial repair could explain the reversal of endothelial function in response to ex

A case of advanced lepromatous leprosy with rhino-oro-laryngological involvement in the post-elimination era.
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A patient presenting with symptoms and signs of congestive heart failure was incidentally found to be in advanced stage of leprosy. He had multiple lepromatous nodules over the entire body. The oral

Acetylcholine as an age-dependent non-neuronal source in the heart.
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In the heart acetylcholine (ACh) slows pacemaker activity depresses contractility and slows conduction in the atrioventricular node. Beside these cardiovascular effects ACh has also been associated w

AAV Serotype-1 Mediates Early Onset of Gene Expression in Mouse Hearts and Results in betterTherapeutic Effect
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PowerPoint Slide Presentation on This paper was published on Gene Therapy (2006) 13, 1495–1502. We studied gene transduciton efficiency mediated by AAV serotypes 1-5 and found AAV serotype 1 is the m

A Cardiac Rehabilitation Program to Improve Psychosocial Outcomes of Women with Heart Disease
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PowerPoint Presentation on A Cardiac Rehabilitation Program to Improve Psychosocial Outcomes of Women with Heart Disease or PowerPoint Presentation on This paper describes the development and impleme

Association between a serotonin transporter gene variant and hopelessness among men in the heart and soul study.
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BACKGROUND Hopelessness is associated with mortality in patients with cardiac disease even after accounting for severity of depression. We sought to determine whether a polymorphism in the promoter r

Novel quinolizidinyl derivatives as antiarrhythmic agents 2. Further investigation.
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Fifteen quinolizidine derivatives have been tested for antiarrhythmic inotropic and chronotropic effects on isolated guinea pig (gp) heart tissues and to assess calcium antagonist activity. All compo

Familial hypertrophic cardiomyopathy can be characterized by a specific pattern of orientation fluctuations of actin molecules .
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A single-point mutation in the gene encoding the ventricular myosin regulatory light chain (RLC) is sufficient to cause familial hypertrophic cardiomyopathy (FHC). Most likely the underlying cause of

Allopurinol in dermatology.
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Off-label use is common in dermatology and is inevitable for rare cutaneous diseases such as perforating dermatosis. Allopurinol is traditionally considered to be a drug for hyperuricemia only but th
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